News
MTORC1 determines autophagy through ULK1 regulation in skeletal muscle.
Castets P, Rüegg MA. Autophagy. 2013 Jul 23;9(9). [Epub ahead of print]
01 Juli 2013
Quinine sulfate as a therapeutic option in a patient with slow channel congenital myasthenic syndrome.
Peyer AK, Abicht A, Heinimann K, Sinnreich M, Fischer D. Neuromuscul Disord. 2013 Jul;23(7):571-4. doi: 10.1016/j.nmd.2013.04.001. Epub 2013 May 18.
01 Juli 2013
During post-natal human myogenesis, normal myotube size requires TRPC1- and TRPC4-mediated Ca²⁺ entry.
Antigny F, Koenig S, Bernheim L, Frieden M.:J Cell Sci. 2013 Jun 1;126(Pt 11):2525-33.
01 Juni 2013
The cranio-sacral progression of muscle development influences the emergence of neuromuscular junction alterations in a severe murine model for Spinal Muscular Atrophy.
Voigt T, Neve A, Schümperli D. Neuropathol Appl Neurobiol. 2013 May 30.[Epub ahead of print]
30 Mai 2013
Sustained activation of mTORC1 in skeletal muscle inhibits constitutive and starvation-induced autophagy and causes a severe, late-onset myopathy.
Castets P, Lin S et al. Cell Metab. 2013 May 7;17(5):731-44. Epub 2013 Apr 18.
07 Mai 2013
Skeletal muscle PGC-1α controls whole-body lactate homeostasis through estrogen-related receptor α-dependent activation of LDH B and repression of LDH A.
Summermatter S, Santos G, Pérez-Schindler J, Handschin C. Proc Natl Acad Sci U S A. 2013 May 21;110(21):8738-43. Epub 2013 May 6.
06 Mai 2013
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